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FAM53B
SNP Table
| variant | genotype | frequency | risk allele |
|---|---|---|---|
| rs139076928 | |||
| rs147886380 | |||
| rs11245332 | |||
| rs1006368 | |||
| rs147832040 | |||
| rs41307542 | |||
| rs61870535 | |||
| rs17624662 | |||
| rs17635335 | |||
| rs3781455 | |||
| rs876352 | |||
| rs4962681 | |||
| rs7917674 | |||
| rs61870502 | |||
| rs200397847 | |||
| rs575229007 | |||
| rs10794173 | |||
| rs10901796 | |||
| rs10901799 | |||
| rs11245333 |
Find on This Page
Summary
The FAM53B gene stimulates the Wnt/beta-catenin pathway, which is crucial for hair growth. Specific gene variants may reduce FAM53B activity. They’ve been associated with relatively higher odds of male-pattern baldness.
Protein names
Found in These Blog Articles
More Information
The FAM53B gene enables the function of beta-catenin, a protein that stimulates hair follicles and promotes hair growth. This protein partakes in a so-called Wnt/beta-catenin pathway, which is often suppressed in people with male pattern baldness [R, R, R, R].
Substances that activate beta-catenin may boost hair growth by reducing the negative impact of excess DHT, which is the culprit behind male-pattern baldness. Researchers are developing new treatment options for this condition that target the Wnt/beta-catenin pathway [R, R].
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